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[9] The mechanism appears to involve direct GHS-R1a receptor activation on cardiac cells independent of GH release, since hexarelin (a related GHRP) showed cardioprotective effects even in GH-deficient models

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This result shows that the amine group has higher electrostatic interactions with bacterial membranes and facilitates the internalization of NPs through membrane penetration

Sex differences in cardiovascular effectiveness of newer glucose-lowering agents: a systematic review