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doi: 10.1007/s11938-019-00231-8

The proposed mechanisms by which hyperhomocysteinaemia can cause harm are common to several conditions

Outside of these causes, it is also possible to develop bursitis from a blow or impact to the affected area

These nucleotides, in response to mechanical stress in the airways activate P2Y2 and A2b receptors on ciliated cells, altering the sodium and chloride composition of the periciliary layer, leading to increased water movement into the lumen which facilitates mucus movement [28, 45, 49, 50]

Breaking this cycle at any point can theoretically resolve a flare, but sustainable improvement requires addressing the cycle at multiple points simultaneously, or finding an intervention that modulates the central controlling mechanisms like NF-kB