Data availability about the pathophysiological processes involved in triggering NIHL has allowed researchers to use compounds, that could act as effective therapies, by targeting specific mechanisms such as the excess generation of free radicals and blood flow restriction to the cochlea
What conversation are we supposed to have with patients
Based on these results, Sung-Jin et al hypothesized that stabilin-1 and stabilin-2 could play an important role in hepatic sequestration of PS-exposed RBCs, suggesting the potential mechanism for the clearance of damaged RBCs by Kupffer cells [273]
This approach also minimizes rebound nausea, headaches, and energy fluctuations some patients experience with abrupt stops
PubMed Shen ZY, Chen YR, Wang MC, Chang SS