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Mechanistic analyses revealed that bacterial inactivation was associated to hydroxyl radical generation, and electrostatic interactions between positively charged N-CQDs and negatively charged bacterial membranes, leading to morphological changes and cell wall damage [133]

Specifically, intra-articular injection or systemic administration of specific ferroptosis inhibitors such as ferrostatin-1, or pharmacologically for example, using natural products or genetically for example, using overexpression vectors upregulating GPX4 expression, activating SLC7A11, or the key transcription factor Nrf2, can, to varying degrees, restore cartilage morphology, reduce chondrocyte death, and significantly lower the expression levels of ECM degradation markers in OA animal models

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Laboratory studies have examined how Epithalon exposure affects gene expression patterns in various cell types