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Indeed, signaling via bile acids can contribute to liver regeneration response following APAP hepatotoxicity as both cholic acid and FGF19 (a downstream mediator of bile acid signaling) treatment result in improved liver regeneration after APAP overdose in mice [31,32]

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To better explain the association between PD dysfunction and metabolic changes in brain regions, several PD-related patterns have been derived

These changes compromise the biomechanical function of the meniscus and reduce its capacity to distribute load, thereby increasing stress on adjacent cartilage and subchondral bone

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