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The core mechanism of action of IGF-1 LR3 IGF-1 LR3 activates downstream signaling pathways (such as the AKT/mTOR pathway) by binding to the IGF-1 receptor (IGF1R) on the cell surface, regulating cell proliferation, differentiation, migration, and metabolism
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210,211 Previous studies have implicated the blockade of CPT1 in the progression of atherosclerosis in macrophages due to increased expression of CD36, a scavenger receptor involved in LDL uptake and subsequent lipid accumulation
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